Effect of Nucleus Amygdalae and Gyrus Cinguli Lesions on Dopamine Receptor 2 in Limbic Region in Rats with Schizophrenia Produced by Methamphetamine 杏仁核和扣带回联合毁损对MAP大鼠脑内多巴胺D2受体的影响
Conclusion The epileptic model has been successfully established by kainic acid microinjected into nucleus amygdalae in Wistar male rats, this model is applicable for temporal lobe epilepsy. 结论:采用杏仁核海人酸微量注射方法成功建立了Wistar大鼠点燃癫痫模型,该模型适用于颞叶癫痫的相关研究。
Conclusions Lesion of nucleus amygdalae and cingulum bundle could restrain the haughty expression of dopamine receptor 2 in brim region because methamphetamine employ in rats. 结论杏仁核和扣带回的联合毁损可以抑制使用MAP而诱发的边缘区D2表达的亢进。
Conclusions It is a simple and effective methods to treat the obstinate epilepsy caused by Hydatid disease, which using the stereoscopic directional skill to excise the cyst of the Hydatid disease and the core-destroying method with heat-coagulation at Nucleus amygdalae and the area of Forel-H. 结论采用立体定向技术行囊虫病灶摘除,杏仁核、Forel-H区热凝核毁损是治疗脑囊虫病诱发顽固性癫痫的简单而有效方法之一。
Conclusion: Our results suggested that programmed cell death-promoting mechanisms are activated by SE and induce neuronal necrosis of nucleus amygdalae, and Mg 2+ has protecting effects on the neurons of nucleus amygdalae. 结论:LPC诱导的SE激活了促进程序性细胞死亡的机制,导致杏仁核神经元坏死,而Mg2+对杏仁核神经元具有保护作用。
Establishment and evaluation of epileptic model kindled by kainic acid microinjected into nucleus amygdalae in rats 海人酸杏仁核点燃大鼠癫痫模型的建立和评价
Mechanism of neuronal death in nucleus amygdalae of rats after lithium-pilocarpine induced status epilepticus 锂-匹鲁卡品致痫大鼠杏仁核神经元损伤机制的实验研究